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Human KCNJ1 Stable Cell Line-HEK293

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Cat.No.
CSC-RI0114
Background
Kir1.1 is an inwardly rectifying potassium channel expessed in kidney, skeletal muscle, liver, pancreas, spleen, and the central nervous system. Mutations in Kir1.1 are responsible for Bartter"s syndrome (renal salt loss) and hereditary hypertension with hyperkalemia. Kir1.1 has therapeutic potential in treatment of hypertension.
Growth Properties
Adherent
Morphology
Epithelial
Host Cell
HEK293
Ship
Dry ice
Gene Information
Official Symbol
KCNJ1
Synonyms
KCNJ1; potassium inwardly-rectifying channel, subfamily J, member 1; ATP-sensitive inward rectifier potassium channel 1; Kir1.1; ROMK1; inwardly rectifying K+ channel; inward rectifier K(+) channel Kir1.1; ATP-regulated potassium channel ROM-K; potassium channel, inwardly rectifying subfamily J member 1; ROMK; KIR1.1;
Gene ID
MIM
UniProt ID
P48048
Chromosome Location
11q24
Pathway
Aldosterone-regulated sodium reabsorption, organism-specific biosystem; Aldosterone-regulated sodium reabsorption, conserved biosystem; Gastric acid secretion, organism-specific biosystem; Gastric acid secretion, conserved biosystem; Inwardly rectifying K+ channels, organism-specific biosystem; Neuronal System, organism-specific biosystem; Potassium Channels, organism-specific biosystem;
Function
ATP binding; inward rectifier potassium channel activity; nucleotide binding; voltage-gated ion channel activity;

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