shRNA set against Rat SMUG1 (NM_177934.2)

Name: shRNA set against Rat SMUG1 (NM_177934.2)
SKU: SHH413740
Availability: InStock

For research use only. Not intended for any clinical use.

Cat. No. : SHH413740

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Product Information

Gene Information

Cat. No.	SHH413740
Description	The SureSilencing trade; shRNA Plasmids are designed to specifically knock down the expression of individual genes by RNA interference under either transient (with GFP) or stable transfection (for hygromycin, neomycin or puromycin-resistance) conditions after performance of the appropriate enrichment or selection procedures, respectively. Each vector contains the shRNA under control of the U1 promoter and either the GFP gene, for the enrichment of transiently transfected cells, or the neomycin or puromycin resistance genes, for the selection of stably transfected cells.
Gene Abbr	SMUG1
Gene Name	Smug1 single-strand selective monofunctional uracil DNA glycosylase [ Mus musculus ]
Species	Rat
Report Gene	GFP
Storage	The SureSilencing shRNA Plasmids are shipped on dry ice or cold packs. Store all tubes at -20°C.

Gene Name	Smug1 single-strand selective monofunctional uracil DNA glycosylase [ Mus musculus ]
Gene Symbol	Smug1
Synonyms	C85220; 1200013B09Rik; A930006H09Rik
Gene Description	single-strand selective monofunctional uracil DNA glycosylase
Gene ID	71726
Uni Prot ID	Q6P5C5
m RNA Refseq	NM_027885.3
Protein Refseq	NP_082161.2
Chromosome Location	15 F3; 15
Function	DNA N-glycosylase activity; DNA N-glycosylase activity; DNA binding; hydrolase activity; hydrolase activity, acting on glycosyl bonds; oxidized base lesion DNA N-glycosylase activity; single-strand selective uracil DNA N-glycosylase activity; single-strand selective uracil DNA N-glycosylase activity; single-stranded DNA binding; uracil DNA N-glycosylase activity; uracil DNA N-glycosylase activity; uracil DNA N-glycosylase activity;
Pathway	Base Excision Repair, organism-specific biosystem; Base excision repair, organism-specific biosystem; Base excision repair, conserved biosystem; Base-Excision Repair, AP Site Formation, organism-specific biosystem; Base-free sugar-phosphate removal via the single-nucleotide replacement pathway, organism-specific biosystem; Cleavage of the damaged pyrimidine, organism-specific biosystem; DNA Repair, organism-specific biosystem;

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